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that side Pulsation may be palpably reduced or absent in the common carotid artery in the neck, in the external carotid artery in front of the ear, and in the internal carotid artery in the lateral wall of the pharynx, but these are among the least dependable signs of carotid disease and too vigorous compression risks dislodging plaque material Also, in the presence of a unilateral internal carotid occlusion, compression of the normal common carotid should be avoided because it may precipitate neurologic symptoms Central retinal artery pressure is reduced on the side of a carotid occlusion or severe stenosis; this can be appreciated by gentle pressure on the globe while viewing the vessels emanating from the optic nerve head A diastolic retinal pressure (determined by ophthalmic dynamometry) of less than 20 mmHg usually means that the common or internal carotid artery is occluded These and other tests for assessing carotid ow have been largely supplanted by ultrasound insonation and MRI imaging of the carotid artery, but retinal examination remains highly valuable in that it may demonstrate emboli within retinal arteries, either shiny white or reddish in appearance; this is another important sign of carotid disease (crystalline cholesterol is sloughed from an atheromatous ulcer) Additional signs of carotid occlusion include pulseless arms (as in Takayasu disease, see page 732); faintness on arising from the horizontal position or recurrent loss of consciousness when walking; headache and sometimes ocular, retro-orbital, and neck pain; transient blindness, either unilateral or bilateral; unilateral visual loss or dimness of vision with exercise, after exposure to bright light, or on assuming an upright position; retinal atrophy and pigmentation; atrophy of the iris; heterochromia of the iris (differing coloration on the two sides is a sign of carotid occlusion early in life); leukomas (corneal scars); peripapillary arteriovenous anastomoses in the retinae; optic atrophy; and claudication of jaw muscles The cardinal clinical sign of stenoses, ulcerations, and dissections of the internal carotid artery near its origin from the common carotid artery are TIAs It is a subject of debate whether these are the result of brin platelet emboli or a reduction in blood ow TIAs are discussed further on, but here it can be stated that TIAs and severe stenosis of the vessel represents an important risk factor for stroke. free code 39 barcode font for word Free Medium-Size Code 39 Font Discontinued - IDAutomation
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